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MedXY AI/MedXY News/Tag: Tauopathy
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Iatrogenic Alzheimer Pathology Can Mirror Severe Sporadic Disease After Cadaveric Growth Hormone Exposure
NeurologyAmyloid-βcadaveric growth hormone

Iatrogenic Alzheimer Pathology Can Mirror Severe Sporadic Disease After Cadaveric Growth Hormone Exposure

By MedXY|Apr 1, 2026

Four men exposed in childhood to cadaveric pituitary-derived human growth hormone developed dementia decades later; one autopsied case showed severe Alzheimer-type pathology, including tauopathy, supporting a transmissible iatrogenic Alzhei

Spinal Cord Tau and Protein Copathologies Associated With Chronic Traumatic Encephalopathy: A Detailed Clinical-Pathological Analysis
NeurologyCTEneurodegeneration

Spinal Cord Tau and Protein Copathologies Associated With Chronic Traumatic Encephalopathy: A Detailed Clinical-Pathological Analysis

By MedXY|Mar 11, 2026

This retrospective study identifies a high prevalence of spinal cord p-tau and protein copathologies in patients with Chronic Traumatic Encephalopathy (CTE), suggesting the existence of a ‘trauma-related encephalomyelopathy’ lin

Redefining CTE: New Autopsy Evidence Links Repetitive Head Impact to Extensive Spinal Cord Pathology
NeurologyCTEneurodegeneration

Redefining CTE: New Autopsy Evidence Links Repetitive Head Impact to Extensive Spinal Cord Pathology

By MedXY|Feb 10, 2026

A landmark autopsy study reveals that Chronic Traumatic Encephalopathy (CTE) involves extensive spinal cord pathology, including tau, TDP-43, and amyloid-β deposits. These findings suggest that repetitive head impacts trigger a widespread e

Age and Sex Redefine the Amyloid-to-Tau Transition: Establishing Patient-Centered PET Thresholds for Alzheimer’s Disease
NeurologyAlzheimer's diseaseprecision medicine

Age and Sex Redefine the Amyloid-to-Tau Transition: Establishing Patient-Centered PET Thresholds for Alzheimer’s Disease

By MedXY|Jan 6, 2026

This study establishes patient-centered amyloid PET thresholds for tauopathy onset, revealing that age and sex significantly influence the transition from amyloidosis to tau accumulation. These personalized cut-offs better predict cognitive

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