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Unraveling Mitochondrial Dysfunction in RBM20-Related Dilated Cardiomyopathy: The Role of Dysregulated Ribonucleoprotein Granules
CardiologyRBM20ribonucleoprotein granules

Unraveling Mitochondrial Dysfunction in RBM20-Related Dilated Cardiomyopathy: The Role of Dysregulated Ribonucleoprotein Granules

By MedXY|Sep 19, 2026

This article reviews novel research showing how cytoplasmic mislocalization of RBM20 impairs mitochondrial function, leading to severe dilated cardiomyopathy and highlighting distinct mechanisms between gain- and loss-of-function RBM20 vari

Targeting Acute Myeloid Leukemia through Multi-Functional Small Molecules: Insights from Cheminformatics and Mitochondrial Modulation
Hematology-Oncology
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targeted therapy
Cheminformatics

Targeting Acute Myeloid Leukemia through Multi-Functional Small Molecules: Insights from Cheminformatics and Mitochondrial Modulation

By MedXY|Sep 1, 2026

Cheminformatic screening identified novel small molecules with apoptotic, autophagic, and reductase-inhibitory activities that selectively kill AML cells, induce mitochondrial dysfunction, and synergize with current therapies, highlighting

Mitochondrial Rescue in HFpEF: Elamipretide Restores Skeletal Muscle Performance in a Rat Model
CardiologyelamipretideHFpEF

Mitochondrial Rescue in HFpEF: Elamipretide Restores Skeletal Muscle Performance in a Rat Model

By MedXY|Jun 15, 2026

Elamipretide improved skeletal muscle force, titin phosphorylation, and atrophy in a HFpEF rat model, supporting cardiolipin stabilization as a promising mitochondrial-targeted strategy.

Can a Mitochondrial “Membrane Stabilizer” Restore Muscle Function in HFpEF? New Rat Data on Elamipretide
CardiologyelamipretideHFpEF

Can a Mitochondrial “Membrane Stabilizer” Restore Muscle Function in HFpEF? New Rat Data on Elamipretide

By MedXY|Jun 19, 2026

In a HFpEF rat model, elamipretide improved skeletal muscle force, reduced titin hyperphosphorylation, and prevented atrophy, supporting cardiolipin stabilization as a promising metabolic strategy.

APOE ε4 Allele as an Independent Predictor of Survival in Parkinson’s Disease Patients
NeurologyAPOE ε4Dementia

APOE ε4 Allele as an Independent Predictor of Survival in Parkinson’s Disease Patients

By MedXY|Oct 21, 2025

The APOE ε4 allele significantly reduces survival in Parkinson’s disease patients independently of dementia, linked to mitochondrial dysfunction and ER stress in PD models.

Unraveling Mitochondrial Dysfunction as a Key Pathological Feature in Long COVID Syndrome
Infectious DiseasesBioenergeticslong COVID

Unraveling Mitochondrial Dysfunction as a Key Pathological Feature in Long COVID Syndrome

By MedXY|Oct 5, 2025

Long COVID patients exhibit distinct mitochondrial bioenergetic impairments, with altered ATP synthase function correlating with clinical symptoms and quality of life, implicating mitochondrial dysfunction in its pathogenesis.

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