Targeting the MEK/RTK Axis: A Promising Combination Therapy for KRAS-Mutant Non-Small Cell Lung Cancer
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KRAS mutations, especially non-G12C subtypes, represent a significant challenge in NSCLC targeted therapy due to limited effective options.
MEK inhibitor trametinib induces adaptive resistance via feedback activation of multiple receptor tyrosine kinases (RTKs).
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This article was created using several editorial tools, including AI, as part of the process. Human editors reviewed this content before publication.